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July 31, 2018 | Dr Reza Moazzeni, Cardiologist |
Last Updated: May 24, 2026
Heart rhythm

What is atrial fibrillation?

Published 31 July 2018 · Reviewed 21 May 2026 · Dr Reza Moazzeni, Cardiologist

The most common heart rhythm disorder — what it feels like, why it matters, and how it's treated. A plain-language guide for patients and their families.

Very common Roughly 1 in 3 adults will develop AF in their lifetime. It becomes more likely with age.
Often silent Many people feel nothing. AF is sometimes first picked up by a smartwatch or routine check.
Highly treatable Modern treatment can control symptoms and dramatically reduce the risk of stroke.
In one paragraph

Atrial fibrillation — AF or AFib — is an irregular heart rhythm where the upper chambers of the heart quiver instead of beating in a coordinated way. It's the most common sustained heart rhythm disorder. AF isn't usually life-threatening on its own, but it significantly raises the risk of stroke and, if the heart races for long periods, can weaken the heart muscle. The good news: with the right combination of medications, lifestyle changes, and sometimes a procedure, most people with AF live full, active lives.

What atrial fibrillation actually is

Dr Reza Moazzeni explains atrial fibrillation — what it is, why it happens, and how it's treated. (About 5 minutes.)

A normal heartbeat is an electrical wave that travels in an orderly pattern from the top of the heart to the bottom. In AF, that orderly wave is replaced by chaos in the heart's two upper chambers — the atria.

Instead of contracting cleanly, the atria quiver — hundreds of disorganised electrical signals firing at once. Only some of those signals make it through to the lower chambers (the ventricles), and they arrive at irregular intervals. The result is an irregular, often fast pulse.

A normal resting heart rate sits between 60 and 100 beats per minute. In uncontrolled AF, the heart rate can climb to 120, 150, even 175 beats per minute at rest. When medications keep the rate around or below 100, doctors call this rate-controlled AF — the rhythm is still irregular, but the heart isn't being overworked.

The simple version: the heart's electrical system has gone off-script in the upper chambers, and the lower chambers respond unevenly. The pulse becomes irregular — sometimes fast, sometimes a normal speed, but rarely steady.

Symptoms (and why some people feel nothing)

AF presents very differently from one person to the next. Some people are aware of every irregular beat. Others have no symptoms at all and only discover they're in AF when a smartwatch alerts them, a GP checks their pulse, or — in the worst case — they have a stroke.

Common symptoms

  • Palpitations — an awareness of the heart racing, fluttering, or beating irregularly
  • Shortness of breath, particularly on exertion
  • Fatigue and reduced exercise capacity
  • Light-headedness or dizziness
  • Chest discomfort (less common, and warrants prompt assessment)
  • Increased need to pass urine during an episode

Silent AF — the case for paying attention

Up to a third of people with AF have no symptoms whatsoever. This is called silent or subclinical AF, and it's a particular concern because the stroke risk is the same — but without symptoms, there's nothing prompting the person to see a doctor.

Smartwatch notification alerting the wearer to a possible atrial fibrillation episode, alongside a confirmatory 12-lead ECG showing AF
A patient in her 60s, active and on no medications, received repeated smartwatch alerts flagging an irregular rhythm. A 12-lead ECG at her GP confirmed AF. Without that early alert, her first sign of AF might have been a stroke. (Click to enlarge.)
If your watch flags AF, don't ignore it. Smartwatch detection is good but not perfect — a confirmed diagnosis still requires an ECG performed by a doctor. Take the watch reading and any ECG strip you've saved to your GP for assessment.

What causes AF and who's at risk

AF rarely has a single cause. It usually emerges from a combination of factors that stress and remodel the upper chambers of the heart over years.

The major risk factors

Risk factor Why it matters
Age The single strongest risk factor. AF affects under 2% of people in their 50s but over 15% of people over 80.
High blood pressure Long-standing hypertension enlarges and stiffens the atria, creating the conditions in which AF develops.
Obesity Excess body weight is a major and modifiable driver. Sustained weight loss can reduce AF episodes substantially.
Obstructive sleep apnoea Repeated drops in oxygen during sleep stress the heart. Treating sleep apnoea is one of the most effective AF interventions.
Alcohol Even moderate drinking raises AF risk. Binge drinking can trigger episodes — sometimes called "holiday heart".
Diabetes Diabetes promotes inflammation and fibrosis in heart tissue, increasing AF risk.
Heart conditions Valve disease (especially mitral valve problems), heart failure, and previous heart attack all increase risk.
Overactive thyroid Hyperthyroidism accelerates the heart and can trigger AF. A simple blood test screens for this.
Family history Having a close relative with AF roughly doubles your own risk.

Other contributors include chronic kidney disease, lung diseases such as COPD, and recent major surgery or serious infection. In some people — particularly younger ones — no clear risk factor is found. This is called lone AF.

The reassuring part: the most powerful risk factors — blood pressure, weight, alcohol, sleep apnoea, diabetes — are all modifiable. Addressing them is now considered as important as any medication.

The three types of AF

AF is grouped by how long episodes last and whether the rhythm returns to normal on its own. The category matters because it guides treatment decisions.

Paroxysmal AF

Episodes start and stop on their own, usually within 24 hours and always within 7 days. Between episodes, the heart is in normal rhythm.

This is often the earliest stage. Episodes may be rare at first and become more frequent over time if risk factors aren't addressed.

Persistent AF

Episodes last longer than 7 days and don't resolve without treatment. Medication or an electrical shock procedure (cardioversion) is needed to restore normal rhythm.

Long-standing persistent AF refers to episodes that have continued for more than a year.

Permanent AF

The heart stays in AF and a joint decision has been made — between patient and cardiologist — to stop trying to restore normal rhythm. Treatment then focuses on controlling the heart rate and preventing stroke.

"Permanent" doesn't always mean forever. If circumstances change (for example, after weight loss or treatment of sleep apnoea), some patients revisit rhythm-control options.

Why AF matters: stroke and heart failure

AF itself rarely causes sudden harm. The reason cardiologists take it seriously is what it can lead to.

Stroke

When the atria quiver instead of contracting, blood pools and can form clots — most often in a small pouch called the left atrial appendage. If a clot breaks free, it travels through the bloodstream and can lodge in a brain artery, causing a stroke. People with untreated AF are roughly five times more likely to have a stroke than people without AF, and AF-related strokes tend to be larger and more disabling than other strokes.

This is why blood-thinning medication (anticoagulation) is central to AF management. Your doctor will use a stroke-risk score to decide whether anticoagulation is appropriate — for most people over 65, and for many younger people with risk factors, the answer is yes.

Heart failure

When the heart races for long periods, the heart muscle gradually weakens — a condition called tachycardia-induced cardiomyopathy. The reassuring part: it's usually reversible once the heart rate is controlled. Even in patients with established heart failure, restoring normal rhythm (where possible) can substantially improve symptoms and outcomes.

The biggest gains in AF care over the last decade have come from two things: better blood thinners, and the realisation that controlling rhythm — not just rate — improves how people feel and live.

How AF is diagnosed

AF is diagnosed by recording the heart's electrical activity during an episode. The standard test is an ECG (electrocardiogram) — a short, painless recording with stickers placed on the chest, arms, and legs.

If you have symptoms but the ECG is normal

This is common — episodes are intermittent and may not coincide with the test. Options to capture an episode include:

  • Holter monitor — a small recorder worn for 24 hours to 14 days
  • Event monitor or loop recorder — worn for longer periods, activated when symptoms occur
  • Implantable loop recorder — a small device placed under the skin that records continuously for up to three years
  • Smartwatch ECG — increasingly accepted as a screening tool, with confirmation by a 12-lead ECG

Tests done alongside the ECG

Once AF is confirmed, most patients have:

  • An echocardiogram to look at heart structure, valve function, and pumping strength
  • Blood tests including thyroid function, kidney function, and electrolytes
  • An assessment of stroke and bleeding risk
  • Sleep history and, where appropriate, a referral for sleep study

How AF is treated

Modern AF care follows three pillars — often referred to as the ABC pathway. Each pillar matters; none replaces the others.

A — Anticoagulation (preventing stroke)

For most patients with AF, a blood thinner is the single most important treatment. The choice today is between:

  • Direct oral anticoagulants (DOACs) — apixaban, rivaroxaban, dabigatran, edoxaban. Preferred for most patients. No INR monitoring, fewer food and drug interactions, and lower bleeding risk than warfarin.
  • Warfarin — still used in specific situations such as mechanical heart valves or moderate-to-severe mitral stenosis. Requires regular INR blood tests.

Aspirin is not effective for stroke prevention in AF and is no longer recommended for this purpose.

B — Better symptom control (rate or rhythm)

The aim here is to make you feel well. Two strategies, often used in combination:

  • Rate control — medications such as beta blockers or calcium channel blockers slow the heart rate so the irregular rhythm doesn't make you breathless or tired.
  • Rhythm control — restoring and maintaining normal sinus rhythm. Options include:
    • Electrical cardioversion — a brief, controlled shock under sedation that resets the rhythm
    • Antiarrhythmic medications — such as flecainide, sotalol, or amiodarone
    • Catheter ablation — a procedure that uses heat or cold energy to isolate the electrical triggers in the upper chambers, typically around the pulmonary veins

Recent evidence has shifted practice: in selected patients with symptomatic paroxysmal AF, catheter ablation is now considered a reasonable first-line option rather than a last resort, with better symptom control and fewer recurrences than medication alone.

C — Comorbidities and risk factors

This pillar is sometimes overlooked, but the evidence is now compelling: treating the conditions that drive AF can halve the frequency of episodes and improve the success of every other treatment.

  • Sustained weight loss (in patients with overweight or obesity)
  • Treating obstructive sleep apnoea
  • Tight blood pressure control
  • Reducing or eliminating alcohol
  • Regular moderate exercise (extreme endurance exercise can paradoxically increase risk)
  • Good control of diabetes and thyroid function
Treating AF without addressing the underlying drivers is like bailing out a leaky boat without patching the hole. Medication and procedures work much better when the upstream causes are managed.

Living with AF

An AF diagnosis is unsettling, but the long-term outlook for well-managed AF is generally good. Most people continue to work, travel, and exercise without major restrictions.

Exercise

Regular moderate exercise is encouraged — it actually reduces AF burden. Walking, swimming, cycling, and structured gym programs are all appropriate. Very high-intensity endurance exercise (marathons, long-distance cycling) is one of the few activities that may slightly increase AF risk in some people.

Travel and flying

AF on its own is not a contraindication to flying. Keep your medication with you in hand luggage and stay well hydrated.

Alcohol and caffeine

Alcohol is a clear trigger. Even one or two drinks can provoke episodes in some patients, and reducing alcohol meaningfully reduces AF burden. Caffeine is much less commonly a trigger — most patients tolerate normal coffee intake without problems.

Mental load

Living with an unpredictable rhythm is genuinely stressful, and anxiety and AF often feed each other. If you find the diagnosis is affecting your mood or sleep, raise it with your GP or cardiologist — it's a normal part of the picture and there's help available.

When to seek urgent help

Most AF episodes are not emergencies. But certain symptoms — whether or not you already have an AF diagnosis — need immediate medical attention.

Call 000 (or your local emergency number) immediately if you have: chest pain or pressure, sudden severe shortness of breath, fainting or near-fainting, sudden weakness or numbness on one side of the body, sudden difficulty speaking or understanding speech, or sudden severe headache.

For sustained palpitations that don't settle within an hour, persistent racing heart, or new symptoms that are different from your usual AF, contact your GP or cardiologist promptly — same-day assessment is usually appropriate.

Frequently asked questions

Can atrial fibrillation be cured?

"Cure" is a strong word for AF, but for many patients — particularly those with paroxysmal AF and well-controlled risk factors — catheter ablation can produce long stretches of normal rhythm and dramatically improved symptoms. Even when AF returns, it's usually less frequent and easier to manage. Permanent rhythm restoration is more realistic than it was a decade ago.

Is AF dangerous?

AF itself is rarely immediately dangerous. The main risks come from its complications — stroke and, less commonly, heart failure — both of which can be substantially reduced with appropriate treatment.

Do I have to take blood thinners forever?

For most patients with a meaningful stroke risk, yes — anticoagulation is long-term. The risk of stroke persists even if your rhythm has been restored, because AF can recur silently. The decision is individual and is reviewed periodically with your cardiologist.

My smartwatch keeps flagging AF. What should I do?

Don't ignore it, and don't panic either. Save any ECG strips your watch records and book a GP appointment. A confirmed diagnosis still requires a doctor-performed ECG, but smartwatch alerts have become a genuinely useful first signal — particularly for silent AF.

Will my children get AF?

There is a familial component, particularly for AF that begins earlier in life. Having a parent or sibling with AF roughly doubles your own risk. That said, modifiable factors — blood pressure, weight, alcohol, sleep apnoea — usually matter more than genetics, and addressing them is the most effective form of prevention.

The bottom line

AF is common, often quiet, and treatable. The two questions every patient with AF should be able to answer with their doctor are: am I on the right protection against stroke? and are the underlying drivers — blood pressure, weight, sleep, alcohol — being addressed? Get those two right and most of AF care follows.

If you've been diagnosed with AF or have symptoms suggesting AF, a cardiologist review brings together rhythm assessment, stroke risk, structural heart evaluation, and a tailored treatment plan in one visit.
Evidence & sources

References & further reading

Clinical references
  1. Guideline Van Gelder IC, Rienstra M, Bunting KV, et al. 2024 ESC Guidelines for the management of atrial fibrillation developed in collaboration with the European Association for Cardio-Thoracic Surgery (EACTS). DOI: 10.1093/eurheartj/ehae176 Eur Heart J. 2024;45(36):3314–3414.
  2. Guideline Brieger D, Amerena J, Attia J, et al. National Heart Foundation of Australia and the Cardiac Society of Australia and New Zealand: Australian clinical guidelines for the diagnosis and management of atrial fibrillation 2018. DOI: 10.5694/mja18.00646 Med J Aust. 2018;209(8):356–362.
  3. Trial analysis Kirchhof P, Camm AJ, Goette A, et al. (EAST-AFNET 4 Investigators) Early rhythm-control therapy in patients with atrial fibrillation. DOI: 10.1056/NEJMoa2019422 N Engl J Med. 2020;383(14):1305–1316.
  4. Trial analysis Andrade JG, Wells GA, Deyell MW, et al. (EARLY-AF Investigators) Cryoablation or drug therapy for initial treatment of atrial fibrillation. DOI: 10.1056/NEJMoa2029554 N Engl J Med. 2021;384(4):305–315.
  5. Cohort study Pathak RK, Middeldorp ME, Lau DH, et al. Aggressive risk factor reduction study for atrial fibrillation and implications for the outcome of ablation: the ARREST-AF cohort study. DOI: 10.1016/j.jacc.2014.09.028 J Am Coll Cardiol. 2014;64(21):2222–2231.
Further reading for patients
  1. Patient body Heart Foundation Australia Atrial fibrillation — patient information. heartfoundation.org.au/your-heart/atrial-fibrillation
  2. Patient body HeartRhythm Alliance Australia (StopAfib) Patient resources and support for people living with AF. heartrhythmalliance.org/aa/au
Next step

Concerned about atrial fibrillation?

Dr Reza Moazzeni provides comprehensive AF assessment at Westmead — rhythm evaluation, stroke risk assessment, echocardiogram, and a tailored treatment plan in one visit. A GP referral is required.

Book a Consultation
Dr Reza Moazzeni, Cardiologist
Reviewed by
Dr Reza Moazzeni MD FRACP
Consultant Cardiologist · Heartcare Sydney

Dr Moazzeni is a consultant cardiologist practising in Westmead, Sydney with expertise in preventive cardiology, echocardiography, and cardiovascular risk assessment. He is a Fellow of the Royal Australasian College of Physicians.